Pancreatic stellate cells activated by mutant KRAS-mediated PAI-1 upregulation foster pancreatic cancer progression via IL-8
MIA PaCa-2 Cell Line - an overview | ScienceDirect Topics
Pancreatic stellate cells activated by mutant KRAS-mediated PAI-1 upregulation foster pancreatic cancer progression via IL-8
Vitamin D Receptor-Mediated Stromal Reprogramming Suppresses Pancreatitis and Enhances Pancreatic Cancer Therapy: Cell
Proteome alterations during clonal isolation of established human pancreatic cancer cell lines | SpringerLink
Frontiers | Loss of SMAD4 Is Associated With Poor Tumor Immunogenicity and Reduced PD-L1 Expression in Pancreatic Cancer
Inhibition of Mesothelin as a Novel Strategy for Targeting Cancer Cells | PLOS ONE
Proteome alterations during clonal isolation of established human pancreatic cancer cell lines | SpringerLink
Silencing growth hormone receptor (GHR) inhibited viability and... | Download Scientific Diagram
Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition - ScienceDirect
MIA PaCa-2 - CRL-1420 | ATCC
Human Pancreatic Cell Lines (ATCC) | Bioz | Ratings For Life-Science Research
Hypoxia-induced circRNF13 promotes the progression and glycolysis of pancreatic cancer | Experimental & Molecular Medicine
A repetitive mutation and selection system for bacterial evolution to increase the specific affinity to pancreatic cancer cells | bioRxiv
Morphofunctional analysis of human pancreatic cancer cell lines in 2- and 3-dimensional cultures | Scientific Reports
MIA PaCa-2 pancreatic xenograft tumor model - Hera BioLabs
Solid stress-induced migration is mediated by GDF15 through Akt pathway activation in pancreatic cancer cells | Scientific Reports
miR‑23a suppresses pancreatic cancer cell progression by inhibiting PLK‑1 expression Corrigendum in /10.3892/mmr.2022.12738
Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition
Cells | Free Full-Text | Mechanical Cues, E-Cadherin Expression and Cell “Sociality” Are Crucial Crossroads in Determining Pancreatic Ductal Adenocarcinoma Cells Behavior
High‐grade mesenchymal pancreatic ductal adenocarcinoma drives stromal deactivation through CSF‐1 | EMBO reports
